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Post Info TOPIC: Green Tea and HCV
Emm


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RE: Green Tea and HCV
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Coffee is extremely hard on the liver as ithas a toxic effect on theliver when it filters the coffee. If a person has a comprosmised liver due to cirrhosis then it is a big no-no. If your liver is not scarred nor hardened you can probably handle one per day as long as you drink 2 cups of water straight after to replace the fluids you will lose from drinking it. Coffee is a diuretic. Also, the water assists the liver and kidneys in filitering it. Another point, if you are on diuretics due to ascites then coffee is really hard on the system and extra water is absolutely necessary.

Green tea is great partly because it prevents free radicals from forming and this is good news.



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Green tea is my new best friend.

Coffee too.

"Increased caffeine consumption is associated with reduced hepatic fibrosis."

http://www.ncbi.nlm.nih.gov/pubmed/20034049




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Report Green Tea and HCV by mikesimon , 1 hour ago Tags: green tea, HCV The green tea polyphenol epigallocatechin-3-gallate (EGCG) inhibits hepatitis C virus (HCV) entry. Abstract Hepatitis C virus (HCV) is a major cause of liver cirrhosis and hepatocellular carcinoma. Current antiviral therapy fails to clear infection in a substantial proportion of cases. Drug development is focused on non-structural proteins required for RNA replication. Individuals undergoing orthotopic liver transplantation face rapid and universal reinfection of the graft. Therefore, antiviral strategies targeting the early stages of infection are urgently needed for the prevention of HCV infection. In this study, we identified the polyphenol epigallocatechin-3-gallate (EGCG) as an inhibitor of HCV entry. Green tea catechins, such as EGCG and its derivatives epigallocatechin (EGC), epicatechingallate (ECG) and epicatechin (EC), have been previously found to exert antiviral and anti-oncogenic properties. EGCG had no effect on HCV RNA replication, assembly or release of progeny virions. However, it potently inhibited HCVcc entry into hepatoma cell lines as well as primary human hepatocytes. The effect was independent of the HCV genotype and both infection of cells by extra-cellular virions and cell-to-cell spread were blocked. Pretreatment of cells with EGCG before HCV inoculation did not reduce HCV infection while application of EGCG during inoculation strongly inhibited HCV infectivity. Moreover, treatment with EGCG directly during inoculation strongly inhibited HCV infectivity. Expression levels of all known HCV (co-)receptors were unaltered by EGCG. Finally, we showed that EGCG inhibits viral attachment to the cell, thus disrupting the initial step of HCV cell entry. Conclusion: the green tea molecule EGCG potently inhibits HCV entry and could be part of an antiviral strategy aimed at the prevention of HCV reinfection after liver transplantation. (HEPATOLOGY 2011.). Copyright 2011 American Association for the Study of Liver Diseases.

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Diagnosed 4/13/10; Started Tx 8/13/10; SVR 7/27/11
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